pathogenesis.
cause confluent bronchopneumonia
often unilateral or more prominent on one side than the other
extensive areas of hemorrhagic necrosis and irregular areas of cavitation.
The pleural surface is covered by a thick layer of exudate.
Numerous abscesses occur, containing clusters of staphylococci, leukocytes,
erythrocytes, and necrotic debris.
Rupture of a small subpleural abscess may result in pyopneumothorax, which may erode
into a bronchus, producing a bronchopleural fistula.
Pneumonia due to S. aureus may be
primary (hematogenous) or
secondary after a viral infection such as influenza.
Hematogenous pneumonia may be secondary to septic emboli, right-sided endocarditis,
or the presence of intravascular devices.
Inhalation pneumonia is caused by alterations of mucociliary clearance, leukocyte
dysfunction, or bacterial adherence initiated by a viral infection.
high fever,
abdominal pain,
tachypnea, dyspnea,
localized or diffuse bronchopneumonia
lobar disease.
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